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This means that during postnatal development in diseased retinas, greater excretion of GSSG, into extracellular space where it get hydrolyzed, could lead to an increase in the [GSH]/[GSSG] ratio

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At the level of mitochondrial metabolic reprogramming, TMAO disrupts pyruvate and fatty acid oxidation, impairs electron transport chain function, reduces ATP synthesis, and induces energy metabolism disorders with concurrent ROS accumulation (Makrecka-Kuka et al., 2017)

Oxidized ascorbate (DHA) can be recycled back to ascorbate (AscH ) by 2-electron reduction catalyzed by dehydroascorbate reductase (DHAR), a key enzyme involved in ascorbate recycling (see Fig